Browse AMR Genes
Explore antimicrobial resistance genes from the literature
Explore antimicrobial resistance genes from the literature
two-component system
Overview
| Protein Change | Nucleotide Change | Mechanism | Organism | Resistance To | Database | Validation Status |
|---|---|---|---|---|---|---|
| T136I | - | - | Staphylococcus aureus | vancomycin | Reslit | Candidate |
| N197S | - | - | Staphylococcus aureus | vancomycin | Reslit | Candidate |
| T224I | - | - | Staphylococcus aureus | vancomycin | Reslit | Candidate |
| D148Q | - | - | Staphylococcus aureus | vancomycin | Reslit | Candidate |
| E99K | - | - | Staphylococcus aureus | vancomycin | Reslit | Candidate |
| E117G | - | - | Staphylococcus aureus | vancomycin | Reslit | Candidate |
| I59L | - | - | Staphylococcus aureus | vancomycin | Reslit | Candidate |
| L26F | - | - | Staphylococcus aureus | vancomycin | Reslit | Candidate |
Crosstalk involving two-component systems in Staphylococcus aureus signaling networks.
Mutations in GraSR contribute to resistance against cell-wall-inhibiting antibiotics.
Impact of vancomycin use trend change due to the availability of alternative antibiotics on the prevalence of Staphylococcus aureus with reduced vancomycin susceptibility: a 14-year retrospective study.