Browse AMR Genes
Explore antimicrobial resistance genes from the literature
Explore antimicrobial resistance genes from the literature
rifampin monooxygenase Rox
Overview
The complete genomic sequence of Nocardia farcinica IFM 10152.
The complete genomic sequence of Nocardia farcinica IFM 10152.
The Structure of the Antibiotic Deactivating, N-hydroxylating Rifampicin Monooxygenase.
The study characterizes the structure and function of the rifampicin monooxygenase (RIFMO) from Nocardia farcinica, which deactivates rifampicin by N-hydroxylation, leading to reduced antimicrobial activity.
Mycolicibacterium fortuitum genomic epidemiology, resistome and virulome.
The study identified an intrinsic resistome in Mycolicibacterium fortuitum comprising genes such as aph(3''-Ic), aac(2')-Ib, arr, blaF, erm39, rbpA, rox, and tap, which confer resistance to various antibiotics including macrolides, aminoglycosides, carbapenems, cephalosporins, and rifampicin. Additionally, a class 1 integron carrying qac/sul1 gene cassette was detected in one genome.
Exploring Nocardia's ecological spectrum and novel therapeutic frontiers through whole-genome sequencing: unraveling drug resistance and virulence factors.
The study identified several antimicrobial resistance genes in Nocardia farcinica, including RbpA, mtrA, FAR-1, rox, blaFAR-1, and blaFAR-1_1, which confer resistance to rifampicin, erythromycin, penicillin, and beta-lactam antibiotics.
Whole Genome Sequencing Highlights the Pathogenic Profile in Nocardia Keratitis.
The study identified antibiotic resistance genes (ARGs) in Nocardia strains, revealing resistance to multiple drugs including rifampin and vancomycin. The mce gene family was linked to the pathogenicity and clinical severity of Nocardia keratitis.
Whole Genome Sequencing Highlights the Pathogenic Profile in Nocardia Keratitis.
The study identified antibiotic resistance genes (ARGs) in Nocardia strains, revealing resistance to multiple drugs including rifampin and vancomycin. The mce gene family was linked to the pathogenicity and clinical severity of Nocardia keratitis.
Genomic characteristics, virulence potential, antimicrobial resistance profiles, and phylogenetic insights into Nocardia cyriacigeorgica.
The study identified several antimicrobial resistance genes in Nocardia cyriacigeorgica, including VanSO and VanRO for vancomycin resistance, erm(O)-Irm, srmB, and ermH for macrolide resistance, bla1 and bcl for beta-lactam resistance, CTX-M and KPC for cephalosporin and carbapenem resistance, tetA(58) and tetB(58) for tetracycline resistance, folC and folP for sulfonamide resistance, rbpA and rpoB2 for rifamycin resistance, murA and AbaF for fosfomycin resistance, cmlR for chloramphenicol resistance, and VatF and VatE for streptogramin resistance.
Genomic characteristics, virulence potential, antimicrobial resistance profiles, and phylogenetic insights into Nocardia cyriacigeorgica.
The study identified several antimicrobial resistance genes in Nocardia cyriacigeorgica, including VanSO and VanRO for vancomycin resistance, erm(O)-Irm, srmB, and ermH for macrolide resistance, bla1 and bcl for beta-lactam resistance, CTX-M and KPC for cephalosporin and carbapenem resistance, tetA(58) and tetB(58) for tetracycline resistance, folC and folP for sulfonamide resistance, rbpA and rpoB2 for rifamycin resistance, murA and AbaF for fosfomycin resistance, cmlR for chloramphenicol resistance, and VatF and VatE for streptogramin resistance.
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